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    Browsing by Author "Eid, Ali"

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        Estrogen receptor and the gender bias in Raynaud's phenomenon. 

        Fardoun, Manal; Kobeissy, Firas; Eid, Ali ( Bentham Science Publishers , 2023 , Article)
        Cold-induced vasoconstriction is a normal physiological reaction aiming at redirecting blood into internal more vital body organs [1]. It is mediated by sympathetic release of norepinephrine as well as by local effectors ...
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        Potato-Herb Synergies as Food Designs for Hyperglycemia and Hypertension Management 

        Saleem, Fahad; Eid, Ali Hussein; Shetty, Kalidas ( Wiley-Blackwell , 2011 , Book chapter)
        The incidence of diabetes mellitus and cardiovascular diseases is increasing at a rapid rate globally. Adult on-set type II diabetes, which is closely associated with obesity, is reported to be 90-95% of all diabetic cases ...
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        Renoprotective Effects of Aldose Reductase Inhibitor Epalrestat against High Glucose-Induced Cellular Injury 

        El Gamal, Heba; Eid, Ali Hussein; Munusamy, Shankar ( Hindawi , 2017 , Article)
        Diabetic nephropathy (DN) is the leading cause of end stage renal disease worldwide. Increased glucose flux into the aldose reductase (AR) pathway during diabetes was reported to exert deleterious effects on the kidney. ...
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        Resveratrol-Elicited PKC Inhibition Counteracts NOX-Mediated Endothelial to Mesenchymal Transition in Human Retinal Endothelial Cells Exposed to High Glucose 

        Giordo, Roberta; Nasrallah, Gheyath K.; Posadino, Anna Maria; Galimi, Francesco; Capobianco, Giampiero; Eid, Ali Hussein; Pintus, Gianfranco... more authors ... less authors ( MDPI , 2021 , Article)
        Diabetes-associated long-term hyperglycaemia leads to oxidative stress-mediated fibrosis in different tissues and organs. Endothelial-to-mesenchymal-transition (EndMT) appears to play a role in diabetes-associated fibrotic ...

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        Qatar University Digital Hub is a digital collection operated and maintained by the Qatar University Library and supported by the ITS department

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