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المؤلفHabas, Elmukhtar
المؤلفIbrahim, Ayman R.
المؤلفMoursi, Moaz O.
المؤلفShraim, Bara A.
المؤلفElgamal, Mohamed E.
المؤلفElzouki, Abdel Naser
تاريخ الإتاحة2023-09-03T09:57:17Z
تاريخ النشر2022-05-01
اسم المنشورArab Journal of Gastroenterology
المعرّفhttp://dx.doi.org/10.1016/j.ajg.2022.01.005
الاقتباسHabas, E., Ibrahim, A. R., Moursi, M. O., Shraim, B. A., Elgamal, M. E., & Elzouki, A. N. (2022). Update on hepatorenal Syndrome: Definition, Pathogenesis, and management. Arab Journal of Gastroenterology, 23(2), 125-133.‏
الرقم المعياري الدولي للكتاب16871979
معرّف المصادر الموحدhttps://www.scopus.com/inward/record.uri?partnerID=HzOxMe3b&scp=85132669101&origin=inward
معرّف المصادر الموحدhttp://hdl.handle.net/10576/47188
الملخصHepatorenal syndrome (HRS) is acute kidney injury (AKI) that occurs without evidence of structural abnormalities in the kidneys in patients with liver disease. It is thought to be due to splanchnic vasculature dilatation that is associated with intense increase of renal arteries’ tone, leading to renal cortex ischemia and AKI. Nitric oxide, endotoxins, neurohormonal changes, bacterial infection, high serum bilirubin and bile acids are examples for factors contributing to HRS development. Nevertheless, other unknown factors may have role in HRS pathophysiology. Hence, further discussion and research are needed to clearly understand HRS. Plasma volume restoration and vasoconstrictors are the cornerstone of HRS treatment. Others such as octreotide, noradrenaline, infection control, systemic inflammatory response prevention, shunting, and renal replacement therapy are currently used to manage HRS. Liver or combined liver and kidney transplantation is currently the ultimate cure for HRS. This review was written to help in better understanding the pathogenesis, diagnosis, and treatment options for HRS.
اللغةen
الناشرArab J Gastroenterol
الموضوعCombined liver-kidney transplantation
Hepatorenal syndrome
HRS pathophysiology
Nitric oxide
Splanchnic vasodilatation
العنوانUpdate on hepatorenal Syndrome: Definition, Pathogenesis, and management
النوعArticle
الصفحات125-133
رقم العدد2
رقم المجلد23
dc.accessType Open Access


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